Rustam_Koviazin Или ты травмировался слабо или всурьёз намерен до нового года личный рекорд установить(на соревнованиях каких выступить)Второе более вероятно.
Дело личное, но не оставил бы ты плечевой сустав и все к нему прилегающее в покое хотяб на недели 2-3.Тренер скорей всего постарше тебя будет, а значит чел старой закалки - всё ради лизурьтата, себя преодолеть и пр.Прыкинь, оно тебе надо?
Lomster Да многих людей от пищевых добавок и смесей мутит.Даже если они нормальным производителем сделаны(про левак промолчу)Если чел здоров и не вегетарьянец, то у него обычно и так карнитина достаточно(за счёт поступления с пищей и синтеза в собственной печени(по большей части))
CARNITINE
L-Carnitine (L-b-hydroxy-g -N,N,N-trimethylaminobutyric acid) is a non-essential organic nutrient required for entry of long-chain fatty acids (as acylcarnitine esters) into mitochondria. It also facilitates removal of short-chain organic acids from mitochondria, thereby freeing intramitochondrial coenzyme A to participate in the b-oxidation and tricarboxylic acid cycle pathways. Carnitine is a substrate for carnitine palmitoyltransferases I and II and carnitine acetyltransferase, enzymes that participate in and regulate fatty acid utilization. Carnitine and its esters are transported across the inner mitochondrial membrane specifically by carnitine-acylcarnitine translocase. The D-isomer of carnitine is biologically inactive. Carnitine is concentrated in most tissues of the body, with about 97% in skeletal muscle. L-Carnitine is synthesized from the essential amino acids lysine and methionine. In humans, the last enzymatic reaction in the pathway occurs primarily in liver and kidney, but not in skeletal and heart muscle. The normal rate of carnitine synthesis in humans is 0.16 to 0.48 mg/kg body weight/day.
Deficiencies: Carnitine deficiency occurs as a primary genetic defect of carnitine transport and secondary to a variety of genetic and acquired disorders. These disorders often are associated with fasting hypoglycemia, lethargy and cardiomyopathy. A murine model of systemic carnitine deficiency (the jvs mouse) has been studied in some detail. Reports of nutritional carnitine deficiency are rare, and in the few cases described, other genetic causes were not specifically excluded. Infants are particularly susceptible to carnitine depletion, because the demands of tissue accretion associated with rapid growth exceed the ability of the infant to synthesize carnitine. However, the substantially lower plasma carnitine concentrations (and, presumably, lower tissue carnitine concentrations) do not impair growth rates or other indicators of normal development.
Diet recommendations: Carnitine is not required in the diet of normal humans. Daily intakes of omnivores and strict vegetarians are 20 mg to 200 mg and <1 mg for a 70 kg (154 lb.) person, respectively. Infant formulas (including total parenteral nutrition solutions) that do not contain carnitine (e.g., non-milk-based formulas) should be supplemented with carnitine to the levels found in human milk, 11.3 mg/L (70 mmol/L).
Food sources: Red meats are the best sources of carnitine (50 to 120 mg/100 g). Fish, chicken and milk are good sources (1.6 to 6.4 mg/100 g). Vegetables, fruits, grains, and other plant-derived foods contain very little carnitine (<0.05 mg/100 g). Approximately 65 to 75% of dietary carnitine is absorbed. Unabsorbed carnitine is almost entirely degraded by bacteria in the large intestine. No dietary components are known to impair absorption. There is no known toxicity associated with ingestion from normal dietary components. Supplements that provide more than 3,000 mg (19 mmol) of carnitine per day may cause diarrhea and/or "fish odor" syndrome.
Recent research: Carnitine may have functions in cellular metabolism independent of its role in fatty acid oxidation, such as plasma membrane fatty acid remodeling, gene regulation (modulation of the transcriptional response to triiodothyronine of genes for malic enzyme and fatty acid synthase), and modulation of cytokine concentrations in experimental sepsis and cancer cachexia. Esters of carnitine (acetyl- and propionylcarnitine) may have pharmacological value, by virtue of their antioxidant properties and/or ability to deliver readily oxidizable carbon units to mitochondria, in chronic disorders such as Alzheimer's disease and ischemia-induced myocardial dysfunction in angina pectoris. Carnitine, by virtue of its role in fatty acid oxidation, may diminish modulation of transcription of urea cycle enzymes by long-chain fatty acids, suggesting a mechanism for its ammeliorating effect on experimentally-induced hyperammonemia.
For further information:
Rebouche, C. J. (1998) Carnitine. In: Modern Nutrition in Health and Disease (Shils, M. E., Olson, J. A., Shike, M. & Ross, A. C., eds.), 9th ed., pp. 505-512. Williams & Wilkins, Baltimore, MD.
Tomomura, M., Tomomura, A., Dewan, A. A. M. & Saheki, T. (1996) Long-chain fatty acids suppress the induction of urea cycle enzyme genes by glucocorticoid action. FEBS Lett. 399: 310-312.
Carnitine and its metabolic role
Long chain fats are metabolised by the mitochondria within the cell. Carnitine is essential to this process, because fat by itself cannot penetrate the membrane of the mitochondria. Each molecule of fat has to be transported across the mitochondrial membrane by binding with a molecule of carnitine. After the fat has been metabolised in the mitochondria, and generated the energy rich ATP, the carnitine is again required to transport the waste product out of the mitochondria as an acyl-carnitine. Carnitine was first identified in 1905, although its metabolic role only began to be understood in the 1950s.
Chemically, carnitine is a quatery amine (the same chemical family that includes choline); its full chemical name is (R)-3-carboxy-2-hydroxy-N,N,N-tri-methyl-1-propanaminium hydroxide. Carnitine come in two forms (stereo-isomers), L-carnitine and D-carnitine. Stereo-isomers have exactly the same chemical formula and structure, except that the molecules of one form are a mirror image of the other. It is only L-carnitine which is the active chemical in the metabolic process, whereas D-carnitine has no effect.
Carnitine is rapidly excreted from the body - its half life is estimated at 17 hours. This means that carnitine must be replaced continuously. In the normal human, approximately 75% of the carnitine is obtained directly as a protein in food and the remaining 25% is synthesised by the body from other proteins in food. The dietary sources richest in carnitine are red meats, particularly lamb and beef. Dairy products contain some carnitine but, with the exception of avocados, vegetables and fruit contain little or no carnitine. The dietary intake of carnitine for optimal health is unknown. An unpublished analysis of hospitalised patients in the US showed dietary intake of between 2 milligrams and 300 milligrams daily while on hospital diet.
Types of carnitine
Carnitine is available both as a prescription drug and as a food supplement from health stores. It is important to understand the differences.
A prescription drug can only be marketed with government approval. To achieve this, the drug must be proven in clinical trials, and be manufactured in an approved manner. Nutritional food supplements or health food products, on the other hand, are not subject to the same rigorous requirements. One consequence is that the prescription version is much more expensive
In the case of carnitine, there is a further difference. Carnitine comes in the two stereo-isomer forms, L-carnitine and D-carnitine. It is only the L form which is biologically active. Some health food versions of carnitine are L-carnitine, and some are a mixture of the L- and the D- forms (DL-carnitine). Because it is much easier to make, DL-carnitine is often much cheaper. There is some evidence to suggest that D-carnitine may be harmful; there may also be some safety issues with the quality of some health food carnitine relating to the imported sources of the bulk chemical. The FDA has issued an Import Alert warning about DL-carnitine.
Dosage
Infants and children: The recommended dosage of carnitine is 50 to 100 mg/kg/day (equivalent to 0.5ml/kg/day Carnitor® oral solution). Higher doses should be administered only with caution and only where clinical and biochemical considerations make it seem likely that higher doses will be of benefit. Dosage should start at 50mg/kg/day, and be increased slowly up to a maximum of 3g/day (30ml/day) while assessing tolerance and therapeutic response. Monitoring should include periodic blood chemistries, vital signs, plasma carnitine concentrations, and overall clinical condition. Carnitor® oral solution may be consumed alone or dissolved in drink or other liquid food. Doses should be spaced evenly throughout the day (every three or four hours) preferable during or following meals and should be consumed slowly in order to maximise tolerance.
Adults: The recommended dosage of carnitine is 1 to 3g/day for a 50kg subject (equivalent to 10 to 30ml/day of Carnitor® oral solution). Higher doses should be administered only with caution and only where clinical and biochemical considerations make it seem likely that higher doses will be of benefit. Dosage should start at 1gm/day, (10ml/day), and be increased slowly while assessing tolerance and therapeutic response. Monitoring should include periodic blood chemistries, vital signs, plasma carnitine concentrations, and overall clinical condition.
Side effects
Carnitine appears largely benign, and there are no known counterindications.
Various mild gastrointestinal complaints have been reported during the long term administration of carnitine; these include transient nausea and vomiting, abdominal cramps, and diarrhoea. Sigma Tau suggest that gastrointestinal adverse reactions with Carnitor® oral solution dissolved in liquids might be avoided by a slow consumption of the solution or by a greater dilution.
Dosage with carnitine may also cause a fishy breath odour. This is caused by excess growth of intestinal flora promoted by carnitine in the gut. The odour may be reduced by increasing the bedtime dose and by spreading the other carnitine doses more regularly through the day. Diminishing the carnitine dose should also eliminate the odour, but it may be better to treat with a broad based antibiotic, so that the carnitine level can be maintained.
It is reported that DL-carnitine, which is sometime sold as a nutritional supplement, has been shown to cause a myasthenia (muscle weakness) type syndrome in some patients. This side effect has not been seen with the L-carnitine form, which is the prescription drug.
Other benefits of carnitine
Carnitine is one of the wonder biochemicals of the nineties, and is being recommended as a treatment for many things.
There are a few groups at particular risk of carnitine deficiency, these include chronic kidney failure patients on hemodialysis and patients with liver failure. Even some healthy subgroups may have additional needs for dietary carnitine. These include strict vegetarians, new-borns, pregnant women and women who are nursing infants.
Research suggests a potentially important role for L-carnitine in the treatment and possibly the prevention of some forms of cardiovascular disease. It was reported some time ago that supplemental carnitine can significantly reduce total blood lipids and derivatives (fats and fat like substances), a finding confirmed in recent years by Japanese researchers who reported that giving 900 milligrams per day of oral L-carnitine could notably reduce levels of blood triglycerides, which are among the lipids and derivatives implicated in cardiovascular disease.
Carnitine in the form of acetyl-L-carnitine is also being used as a treatment for Alzheimer’s disease. Acetyl-L-carnitine affects the cholinergic system, the dopaminergic and NMDA receptor systems; all these affect brain activity.
Like MCT oil, carnitine is being touted as a wonder additive by health food stores. Among the many claims for carnitine are that it reduces heart disease, promotes intelligence, slows the ageing process and can be used as a dieting agent. It is also being used as a supplement by athletes to enhance performance, on the basis that it promotes the metabolism of fat, and improves the efficiency of muscles.
Надеюсь, ты справишься с этим сам или при помощи тов Промта
Но так, в принципе, прикольно... перед барышней выпендриться. 
Я, наверно, не смогу. Хотя попробовать просто не на чем. 
Теми же руками открыть стеклянную бутыль с пивом/газировкой с металлической(маленькой) крышечкой. Ясень пень бутылка должна быть запечатана, т.е. до этого ни разу не вскрываться.
. Вот теперь по телефону хочу его поймать, спросить еще раз. 